MYO1F
跳至導覽
跳至搜尋
檢索資訊框的維基數據項目時發生錯誤
MYO1F (化學式為：C5543H8726O1622N1590S39[1])是一個位於人類17號染色體上的基因，該編碼是一種隸屬於肌凝蛋白超家族的蛋白肌凝蛋白-If（Myosin-If）。MYO1F編碼的肌凝蛋白-If是一種非典型的肌凝蛋白，與編碼肌凝蛋白-1MYH1是兩種不同的基因。[2][3][4][5]
MYO1F基因主要在免疫系統中表達，其功能主要與細胞黏附，細胞移動有關[6]。而該基因的突變則與非綜合症性耳聾有關[7]。
參見[編輯]
參考資料[編輯]
- ^ Expasy - ProtParam. web.expasy.org. [2026-02-13].
- ^ MYO1F Gene. GeneCards. [2021-05-02]. （原始內容存檔於2021-06-04）.
- ^ Crozet F, el Amraoui A, Blanchard S, Lenoir M, Ripoll C, Vago P, Hamel C, Fizames C, Levi-Acobas F, Depetris D, Mattei MG, Weil D, Pujol R, Petit C. Cloning of the genes encoding two murine and human cochlear unconventional type I myosins. Genomics. Apr 1997, 40 (2): 332–41. PMID 9119401. doi:10.1006/geno.1996.4526.
- ^ Hasson T, Skowron JF, Gilbert DJ, Avraham KB, Perry WL, Bement WM, Anderson BL, Sherr EH, Chen ZY, Greene LA, Ward DC, Corey DP, Mooseker MS, Copeland NG, Jenkins NA. Mapping of unconventional myosins in mouse and human. Genomics. Feb 1997, 36 (3): 431–9. PMID 8884266. doi:10.1006/geno.1996.0488.
- ^ Entrez Gene: MYO1F myosin IF. [2021-05-02]. （原始內容存檔於2010-12-05）.
- ^ Kim SV, Mehal WZ, Dong X, Heinrich V, Pypaert M, Mellman I, Dembo M, Mooseker MS, Wu D, Flavell RA. Modulation of cell adhesion and motility in the immune system by Myo1f. Science. October 2006, 314 (5796): 136–9. PMID 17023661. doi:10.1126/science.1131920.
- ^ Chen AH, Stephan DA, Hasson T, Fukushima K, Nelissen CM, Chen AF, Jun AI, Ramesh A, Van Camp G, Smith RJ. MYO1F as a candidate gene for nonsyndromic deafness, DFNB15. Arch. Otolaryngol. Head Neck Surg. August 2001, 127 (8): 921–5. PMID 11493199. doi:10.1001/archotol.127.8.921.
拓展閱讀[編輯]
- Bement WM, Hasson T, Wirth JA, et al. Identification and overlapping expression of multiple unconventional myosin genes in vertebrate cell types. Proc. Natl. Acad. Sci. U.S.A. 1994, 91 (14): 6549–53. PMC 44240 可免費查閱. PMID 8022818. doi:10.1073/pnas.91.14.6549.
- Krugmann S, Anderson KE, Ridley SH, et al. Identification of ARAP3, a novel PI3K effector regulating both Arf and Rho GTPases, by selective capture on phosphoinositide affinity matrices. Mol. Cell. 2002, 9 (1): 95–108. PMID 11804589. doi:10.1016/S1097-2765(02)00434-3.
- Strausberg RL, Feingold EA, Grouse LH, et al. Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences. Proc. Natl. Acad. Sci. U.S.A. 2003, 99 (26): 16899–903. PMC 139241 可免費查閱. PMID 12477932. doi:10.1073/pnas.242603899.
- Ota T, Suzuki Y, Nishikawa T, et al. Complete sequencing and characterization of 21,243 full-length human cDNAs. Nat. Genet. 2004, 36 (1): 40–5. PMID 14702039. doi:10.1038/ng1285.
- Gerhard DS, Wagner L, Feingold EA, et al. The Status, Quality, and Expansion of the NIH Full-Length cDNA Project: The Mammalian Gene Collection (MGC). Genome Res. 2004, 14 (10B): 2121–7. PMC 528928 可免費查閱. PMID 15489334. doi:10.1101/gr.2596504.
- Rual JF, Venkatesan K, Hao T, et al. Towards a proteome-scale map of the human protein-protein interaction network. Nature. 2005, 437 (7062): 1173–8. PMID 16189514. doi:10.1038/nature04209.
- Olsen JV, Blagoev B, Gnad F, et al. Global, in vivo, and site-specific phosphorylation dynamics in signaling networks. Cell. 2006, 127 (3): 635–48. PMID 17081983. doi:10.1016/j.cell.2006.09.026.
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