编辑“︁UBTF”︁（章节）

==相互作用==
UBTF已知能与{{le|酪蛋白激酶2α亚基|Casein kinase 2, alpha 1}}<ref name=pmid7651819>{{cite journal |last=Voit |first=R |authorlink= |coauthors=Kuhn A, Sander E E, Grummt I |date=July 1995 |title=Activation of mammalian ribosomal gene transcription requires phosphorylation of the nucleolar transcription factor UBF |journal=Nucleic Acids Res. |volume=23 |issue=14 |pages=2593–9 |publisher= |location = ENGLAND| issn = 0305-1048| pmid = 7651819 | bibcode = | oclc =| id = | url = | language = | format = | accessdate = | laysummary = | laysource = | laydate = | quote = |doi=10.1093/nar/23.14.2593 |pmc=307079 }}</ref>、[[TAF1C]]<ref name=pmid11698641>{{cite journal |last=Voit |first=R |authorlink= |coauthors=Grummt I |date=November 2001 |title=Phosphorylation of UBF at serine 388 is required for interaction with RNA polymerase I and activation of rDNA transcription |journal=[[PNAS|Proc. Natl. Acad. Sci. U.S.A.]] |volume=98 |issue=24 |pages=13631–6 |publisher= |location = United States| issn = 0027-8424| pmid = 11698641 |doi = 10.1073/pnas.231071698 | bibcode = | oclc =| id = | url = | language = | format = | accessdate = | laysummary = | laysource = | laydate = | quote = |pmc=61092 }}</ref><ref name=pmid10913176>{{cite journal |last=Zhai |first=W |authorlink= |coauthors=Comai L |date=August 2000 |title=Repression of RNA Polymerase I Transcription by the Tumor Suppressor p53 |journal=Mol. Cell. Biol. |volume=20 |issue=16 |pages=5930–8 |publisher= |location = UNITED STATES| issn = 0270-7306| pmid = 10913176 | bibcode = | oclc =| id = | url =https://archive.org/details/sim_molecular-and-cellular-biology_2000-08_20_16/page/5930| language = | format = | accessdate = | laysummary = | laysource = | laydate = | quote = |doi=10.1128/MCB.20.16.5930-5938.2000 |pmc=86070 }}</ref>、[[视网膜母细胞瘤蛋白]]<ref name=pmid11042686>{{cite journal |last=Hannan |first=K M |authorlink= |coauthors=Hannan R D, Smith S D, Jefferson L S, Lun M, Rothblum L I |date=October 2000 |title=Rb and p130 regulate RNA polymerase I transcription: Rb disrupts the interaction between UBF and SL-1 |journal=Oncogene |volume=19 |issue=43 |pages=4988–99 |publisher= |location = ENGLAND| issn = 0950-9232| pmid = 11042686 |doi = 10.1038/sj.onc.1203875 | bibcode = | oclc =| id = | url = | language = | format = | accessdate = | laysummary = | laysource = | laydate = | quote = }}</ref>和[[TAF1]]<ref name=pmid12498690>{{cite journal |last=Lin |first=Chih-Yin |authorlink= |coauthors=Tuan JoAnn, Scalia Pierluigi, Bui Tiffany, Comai Lucio |date=Dec 2002 |title=The cell cycle regulatory factor TAF1 stimulates ribosomal DNA transcription by binding to the activator UBF |journal=Curr. Biol. |volume=12 |issue=24 |pages=2142–6 |publisher= |location = England| issn = 0960-9822| pmid = 12498690 | bibcode = | oclc =| id = | url = | language = | format = | accessdate = | laysummary = | laysource = | laydate = | quote = |doi=10.1016/S0960-9822(02)01389-1 }}</ref>进行[[蛋白質交互作用|交互作用]]。